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This decrease is thought to represent an adaptive response to reduced carbohydrate availability, as lower insulin and glucose levels impair the activity of deiodinase enzymes (DIO) that convert T4 to active T3 in peripheral tissues [13]

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Notably, NF-κB can be activated independently of TLRs by inflammatory cytokines, such as Tumor Necrosis Factor-alpha (TNF-α), which are abundant in the tumor microenvironment