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Copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of milk protein and lipid in bovine mammary epithelial cells - ScienceDirect

Notably, metabolite activity is attenuated in these backgrounds, indicating that UPS loss-of-function epistatically regulates metabolite activity and suggests that UPS-linked protein homeostasis defects may result from metabolite exposure

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These injection sites have nerves and blood vessels