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The JNK pathway also plays a role in the resolution of inflammation by promoting apoptosis in dysfunctional cells, thereby limiting excessive inflammation (161), and by promoting autophagy which reduces the debris-mediated inflammation (161, 162)
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Prolactin: Similarly, ipamorelin's reduced propensity to stimulate prolactin compared to GHRP-2 and GHRP-6 is a pharmacological distinction, not a guarantee
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